Education
PhD, University of Padova, Italy
Current position
Post Doc, Biomedical Sciences, University of Padova, Italy
Publications
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Zanetti, G., Sikorra, S., Rummel, A., Krez, N., Duregotti, E., Negro, S., Henke, T., Rossetto, O., Binz, T. and Pirazzini, M. (2017). Botulinum neurotoxin C mutants reveal different effects of syntaxin or SNAP-25 proteolysis on neuromuscular transmission. PLoS Pathog 13(8): e1006567.
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Negro, S., Lessi, F., Duregotti, E., Aretini, P., La Ferla, M., Franceschi, S., Menicagli, M., Bergamin, E., Radice, E., Thelen, M., Megighian, A., Pirazzini, M., Mazzanti, C. M., Rigoni, M. and Montecucco, C. (2017). CXCL12alpha/SDF-1 from perisynaptic Schwann cells promotes regeneration of injured motor axon terminals. EMBO Mol Med 9(8): 1000-1010.
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Rodella, U., Negro, S., Scorzeto, M., Bergamin, E., Jalink, K., Montecucco, C., Yuki, N. and Rigoni, M. (2017). Schwann cells are activated by ATP released from neurons in an in vitro cellular model of Miller Fisher syndrome. Dis Model Mech 10(5): 597-603.
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Rodella, U., Scorzeto, M., Duregotti, E., Negro, S., Dickinson, B. C., Chang, C. J., Yuki, N., Rigoni, M. and Montecucco, C. (2016). An animal model of Miller Fisher syndrome: Mitochondrial hydrogen peroxide is produced by the autoimmune attack of nerve terminals and activates Schwann cells. Neurobiol Dis 96: 95-104.
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Negro, S., Bergamin, E., Rodella, U., Duregotti, E., Scorzeto, M., Jalink, K., Montecucco, C. and Rigoni, M. (2016). ATP Released by Injured Neurons Activates Schwann Cells. Front Cell Neurosci 10: 134.
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Duregotti, E., Negro, S., Scorzeto, M., Zornetta, I., Dickinson, B. C., Chang, C. J., Montecucco, C. and Rigoni, M. (2015). Mitochondrial alarmins released by degenerating motor axon terminals activate perisynaptic Schwann cells. Proc Natl Acad Sci U S A 112(5): E497-505.