Education

Bachelor of Science, Houston Baptist University, Houston, Texas USA, 1991

Current position

Research Associate, Pediatric Surgery, University of Texas Health Science Center, Houston, Texas USA

Publications

1. Lee, H. J., Diaz, M. F., Ewere, A., Olson, S. D., Cox, C. S., Jr. and Wenzel, P. L. (2017). Focal adhesion kinase signaling regulates anti-inflammatory function of bone marrow mesenchymal stromal cells induced by biomechanical force. Cell Signal 38: 1-9.
2. Diaz, M. F., Vaidya, A. B., Evans, S. M., Lee, H. J., Aertker, B. M., Alexander, A. J., Price, K. M., Ozuna, J. A., Liao, G. P., Aroom, K. R., Xue, H., Gu, L., Omichi, R., Bedi, S., Olson, S. D., Cox, C. S., Jr. and Wenzel, P. L. (2017). Biomechanical Forces Promote Immune Regulatory Function of Bone Marrow Mesenchymal Stromal Cells. Stem Cells 35(5): 1259-1272.
3. Lee, H. J., Diaz, M. F., Price, K. M., Ozuna, J. A., Zhang, S., Sevick-Muraca, E. M., Hagan, J. P. and Wenzel, P. L. (2017). Fluid shear stress activates YAP1 to promote cancer cell motility. Nat Commun 8: 14122.
4. Diaz, M. F., Li, N., Lee, H. J., Adamo, L., Evans, S. M., Willey, H. E., Arora, N., Torisawa, Y., Vickers, D. A., Morris, S. A., Naveiras, O., Murthy, S. K., Ingber, D. E., Daley, G. Q., García-Cardeña, G. and Wenzel, P. L. (2015). Biomechanical Forces Promote Blood Development through Prostaglandin E2 and the cAMP-PKA Signaling Axis. J Exp Med 212: 665-680.
5. Li, N., Diaz, M. F. and Wenzel, P. L. (2015). Application of fluid mechanical force to embryonic sources of hemogenic endothelium and hematopoietic stem cells. Methods Mol Biol 1212: 183-193.
6. Lee, H. J., Li, N., Evans, S. M., Diaz, M. F. and Wenzel, P. L. (2013). Biomechanical force in blood development: extrinsic physical cues drive pro-hematopoietic signaling. Differentiation 86(3): 92-103.
7. Wang, Y., Barker, K., Shi, S., Diaz, M., Mo, B. and Gutstein, H. B. (2012). Blockade of PDGFR-beta activation eliminates morphine analgesic tolerance. Nat Med 18(3): 385-387.
8. Mouledous, L., Diaz, M. F. and Gutstein, H. B. (2007). Extracellular signal-regulated kinase (ERK) inhibition does not prevent the development or expression of tolerance to and dependence on morphine in the mouse. Pharmacol Biochem Behav 88(1): 39-46.
9. Xu, J. J., Walla, B. C., Diaz, M. F., Fuller, G. N. and Gutstein, H. B. (2006). Intermittent lumbar puncture in rats: a novel method for the experimental study of opioid tolerance. Anesth Analg 103(3): 714-720.
10. Mouledous, L., Diaz, M. F. and Gutstein, H. B. (2004). Modulation of extracellular signal-regulated kinase (ERK) activity by acute and chronic opioid treatment in neuronal and glial cell lines. J Neurochem 90(6): 1371-1377.
11. Fraizer, G., Leahy, R., Priyadarshini, S., Graham, K., Delacerda, J. and Diaz, M. (2004). xFraizer, G., Leahy, R., Priyadarshini, S., Graham, K., Delacerda, J. and Diaz, M. (2004). Suppression of prostate tumor cell growth in vivo by WT1, the Wilms' tumor suppressor gene. Int J Oncol 24(3): 461-471. Int J Oncol 24(3): 461-471.
12. Andreopoulos, D., Kasi, L. P., Kim, E. E., Diaz, M., Yang, D. J. and Asimacopoulos, P. J. (2002). Detection of Acute Postoperative Mediastinitis in mice Using Tc99m-liposomes. Invest Rad 37(8): 435-439.
13. Diaz, M., Abdul, M. and Hoosein, N. (1998). Modulation of neuroendocrine differentiation in prostate cancer by interleukin-1 and -2. Prostate Suppl 8: 32-36.
14. Diaz, M., Macey, D. J., Kasi, L. P., Bayouth, J. E. and Podoloff, D. A. (1995). Dispensing TherapeuticAmounts of Holmium-166 with a Radionuclide Calibrator. J Nucl Med Tech 23(4): 275-278.