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Clara Lubeseder-Martellato

Education

Ph.D., Helmholtz Zentrum Munich (former GSF), Germany, 2003

Current position

Postdoc (part-time), II Medical dep., Klinikum Rechts der Isar, TU Munich, Germany

Publications

  1. Ardito, C. M., Gruner, B. M., Takeuchi, K. K., Lubeseder-Martellato, C., Teichmann, N., Mazur, P. K., Delgiorno, K. E., Carpenter, E. S., Halbrook, C. J., Hall, J. C., Pal, D., Briel, T., Herner, A., Trajkovic-Arsic, M., Sipos, B., Liou, G. Y., Storz, P., Murray, N. R., Threadgill, D. W., Sibilia, M., Washington, M. K., Wilson, C. L., Schmid, R. M., Raines, E. W., Crawford, H. C. and Siveke, J. T. (2012). EGF receptor is required for KRAS-induced pancreatic tumorigenesis. Cancer Cell 22(3): 304-317.
  2. Heid, I., Lubeseder-Martellato, C. (equally contributed), Sipos, B., Mazur, P. K., Lesina, M., Schmid, R. M. and Siveke, J. T. (2011). Early requirement of Rac1 in a mouse model of pancreatic cancer. Gastroenterology 141(2): 719-730, 730 e711-717.
  3. Siveke, J. T., Lubeseder-Martellato, C., Lee, M., Mazur, P. K., Nakhai, H., Radtke, F. and Schmid, R. M. (2008). Notch signaling is required for exocrine regeneration after acute pancreatitis. Gastroenterology 134(2): 544-555.
  4. Siveke, J. T., Einwachter, H., Sipos, B., Lubeseder-Martellato, C., Kloppel, G. and Schmid, R. M. (2007). Concomitant pancreatic activation of KrasG12D and Tgfa results in cystic papillary neoplasms reminiscent of human IPMN. Cancer Cell 12(3): 266-279.
  5. Naschberger, E., Lubeseder-Martellato, C. (equally contributed), Meyer, N., Gessner, R., Kremmer, E., Gessner, A. and Sturzl, M. (2006). Human guanylate binding protein-1 is a secreted GTPase present in increased concentrations in the cerebrospinal fluid of patients with bacterial meningitis. Am J Pathol 169(3): 1088-1099.
  6. Naschberger, E., Werner, T., Vicente, A. B., Guenzi, E., Topolt, K., Leubert, R., Lubeseder-Martellato, C., Nelson, P. J. and Sturzl, M. (2004). Nuclear factor-kappaB motif and interferon-alpha-stimulated response element co-operate in the activation of guanylate-binding protein-1 expression by inflammatory cytokines in endothelial cells. Biochem J 379(Pt 2): 409-420.
  7. Guenzi, E., Topolt, K., Lubeseder-Martellato, C., Jorg, A., Naschberger, E., Benelli, R., Albini, A. and Sturzl, M. (2003). The guanylate binding protein-1 GTPase controls the invasive and angiogenic capability of endothelial cells through inhibition of MMP-1 expression. EMBO J 22(15): 3772-3782.
  8. Lubeseder-Martellato, C. (equally contributed), Guenzi, E., Jorg, A., Topolt, K., Naschberger, E., Kremmer, E., Zietz, C., Tschachler, E., Hutzler, P., Schwemmle, M., Matzen, K., Grimm, T., Ensoli, B. and Sturzl, M. (2002). Guanylate-binding protein-1 expression is selectively induced by inflammatory cytokines and is an activation marker of endothelial cells during inflammatory diseases. Am J Pathol 161(5): 1749-1759.
  9. Guenzi, E., Topolt, K., Cornali, E., Lubeseder-Martellato, C. equally contributed), Jorg, A., Matzen, K., Zietz, C., Kremmer, E., Nappi, F., Schwemmle, M., Hohenadl, C., Barillari, G., Tschachler, E., Monini, P., Ensoli, B. and Sturzl, M. (2001). The helical domain of GBP-1 mediates the inhibition of endothelial cell proliferation by inflammatory cytokines. EMBO J 20(20): 5568-5577.
Protocols by Clara Lubeseder-Martellato
  1. Isolation, Culture and Differentiation of Primary Acinar Epithelial Explants from Adult Murine Pancreas