Search

Reviewer
Laura Campisi
  • Icahn School of Medicine at Mount Sinai
Research focus
  • Immunology
  • 1 Author merit

Education

Ph.D in Immunology, University of Nice-Sophia Antipolis, France, 2009

Current position

Instructor, Microbiology, Icahn School of Medicine at Mount Sinai, New York, USA

Publications

  1. Campisi, L., Barbet, G., Ding, Y., Esplugues, E., Flavell, R. A. and Blander, J. M. (2016). Apoptosis in response to microbial infection induces autoreactive TH17 cells. Nat Immunol 17(9): 1084-1092.
  2. Rialdi, A., Campisi, L., Zhao, N., Lagda, A. C., Pietzsch, C., Ho, J. S., Martinez-Gil, L., Fenouil, R., Chen, X., Edwards, M., Metreveli, G., Jordan, S., Peralta, Z., Munoz-Fontela, C., Bouvier, N., Merad, M., Jin, J., Weirauch, M., Heinz, S., Benner, C., van Bakel, H., Basler, C., Garcia-Sastre, A., Bukreyev, A. and Marazzi, I. (2016). Topoisomerase 1 inhibition suppresses inflammatory genes and protects from death by inflammation. Science 352(6289): aad7993.
  3. Heaton, N. S., Moshkina, N., Fenouil, R., Gardner, T. J., Aguirre, S., Shah, P. S., Zhao, N., Manganaro, L., Hultquist, J. F., Noel, J., Sachs, D., Hamilton, J., Leon, P. E., Chawdury, A., Tripathi, S., Melegari, C., Campisi, L., Hai, R., Metreveli, G., Gamarnik, A. V., Garcia-Sastre, A., Greenbaum, B., Simon, V., Fernandez-Sesma, A., Krogan, N. J., Mulder, L. C., van Bakel, H., Tortorella, D., Taunton, J., Palese, P. and Marazzi, I. (2016). Targeting Viral Proteostasis Limits Influenza Virus, HIV, and Dengue Virus Infection. Immunity 44(1): 46-58.
  4. Campisi, L., Cummings, R. J. and Blander, J. M. (2014). Death-defining immune responses after apoptosis. Am J Transplant 14(7): 1488-1498.
  5. Blander, J. M., Torchinsky, M. B. and Campisi, L. (2012). Revisiting the old link between infection and autoimmune disease with commensals and T helper 17 cells. Immunol Res 54(1-3): 50-68.
  6. Campisi, L., Soudja, S. M., Cazareth, J., Bassand, D., Lazzari, A., Brau, F., Narni-Mancinelli, E., Glaichenhaus, N., Geissmann, F. and Lauvau, G. (2011). Splenic CD8alpha(+) dendritic cells undergo rapid programming by cytosolic bacteria and inflammation to induce protective CD8(+) T-cell memory. Eur J Immunol 41(6): 1594-1605.
  7. Rahmoun, M., Gros, M., Campisi, L., Bassand, D., Lazzari, A., Massiera, C., Narni-Mancinelli, E., Gounon, P. and Lauvau, G. (2011). Priming of protective anti-Listeria monocytogenes memory CD8+ T cells requires a functional SecA2 secretion system. Infect Immun 79(6): 2396-2403.
  8. Bougneres, L., Helft, J., Tiwari, S., Vargas, P., Chang, B. H., Chan, L., Campisi, L., Lauvau, G., Hugues, S., Kumar, P., Kamphorst, A. O., Dumenil, A. M., Nussenzweig, M., MacMicking, J. D., Amigorena, S. and Guermonprez, P. (2009). A role for lipid bodies in the cross-presentation of phagocytosed antigens by MHC class I in dendritic cells. Immunity 31(2): 232-244.
  9. Auffray, C., Fogg, D. K., Narni-Mancinelli, E., Senechal, B., Trouillet, C., Saederup, N., Leemput, J., Bigot, K., Campisi, L., Abitbol, M., Molina, T., Charo, I., Hume, D. A., Cumano, A., Lauvau, G. and Geissmann, F. (2009). CX3CR1+ CD115+ CD135+ common macrophage/DC precursors and the role of CX3CR1 in their response to inflammation. J Exp Med 206(3): 595-606.
  10. Geissmann, F., Auffray, C., Palframan, R., Wirrig, C., Ciocca, A., Campisi, L., Narni-Mancinelli, E. and Lauvau, G. (2008). Blood monocytes: distinct subsets, how they relate to dendritic cells, and their possible roles in the regulation of T-cell responses. Immunol Cell Biol 86(5): 398-408.
  11. Campisi, L., Brau, F. and Glaichenhaus, N. (2008). Visualisation of immune responses against pathogens. Immunol Rev 221:188-199.
  12. Narni-Mancinelli, E., Campisi, L., Bassand, D., Cazareth, J., Gounon, P., Glaichenhaus, N. and Lauvau, G. (2007). Memory CD8+ T cells mediate antibacterial immunity via CCL3 activation of TNF/ROI+ phagocytes. J Exp Med 204(9): 2075-2087.
1 Protocol published
In vitro Antigen-presentation Assay for Self- and Microbial-derived Antigens
Author:  Laura Campisi, date: 06/05/2017, view: 1322, Q&A: 0
Antigen presenting cells (APC) are able to process and present to T cells antigens from different origins. This mechanism is highly regulated, in particular by Patter Recognition Receptor (PRR) signals. Here, I detail a protocol designed to assess ...